Search results for " Graves' ophthalmopathy"

showing 3 items of 3 documents

Evidence That Graves' Ophthalmopathy Immunoglobulins Do Not Directly Activate IGF-1 Receptors

2018

Background: Graves' ophthalmopathy (GO) pathogenesis involves thyrotropin (TSH) receptor (TSHR)-stimulating autoantibodies. Whether there are autoantibodies that directly stimulate insulin-like growth factor 1 receptors (IGF-1Rs), stimulating insulin-like growth factor receptor antibodies (IGFRAbs), remains controversial. This study attempted to determine whether there are stimulating IGFRAbs in patients with GO. Methods: Immunoglobulins (Igs) were purified from normal volunteers (NV-Igs) and patients with GO (GO-Igs). The effects of TSH, IGF-1, NV-Igs, and GO-Igs on pAKT and pERK1/2, members of pathways used by IGF-1R and TSHR, were compared in orbital fibroblasts from GO patients (GOFs) a…

0301 basic medicineendocrine systemendocrine system diseasesEndocrinology Diabetes and MetabolismImmunoglobulins030209 endocrinology & metabolismCell LineReceptor IGF Type 1PathogenesisGraves' ophthalmopathy03 medical and health sciences0302 clinical medicineEndocrinologymedicineHumansImmunology Autoimmunity and Graves' OphthalmopathyPhosphorylationReceptorbiologybusiness.industryAutoantibodyReceptors Thyrotropinmedicine.diseaseeye diseasesGraves Ophthalmopathy030104 developmental biologyImmunologybiology.proteinlipids (amino acids peptides and proteins)AntibodybusinessProto-Oncogene Proteins c-akthormones hormone substitutes and hormone antagonists
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Graves' Autoantibodies Exhibit Different Stimulating Activities in Cultures of Thyrocytes and Orbital Fibroblasts Not Reflected by Clinical Assays

2021

Background: The pathogenesis of Graves' hyperthyroidism (GH) and associated Graves' orbitopathy (GO) appears to involve stimulatory autoantibodies (thyrotropin receptor [TSHR]-stimulating antibodies [TSAbs]) that bind to and activate TSHRs on thyrocytes and orbital fibroblasts. In general, measurement of circulating TSHR antibodies by clinical assays correlates with the status of GH and GO. However, most clinical measurements of TSHR antibodies use competitive binding assays that do not distinguish between TSAbs and antibodies that bind to but do not activate TSHRs. Moreover, clinical assays for TSAbs measure stimulation of only one signaling pathway, the cyclic adenosine monophosphate (cAM…

AdultMaleendocrine systemmedicine.medical_specialtyendocrine system diseasesEndocrinology Diabetes and Metabolismmedicine.medical_treatmentGraves' diseaseThyrotropinStimulationEndocrinologyimmune system diseasesInternal medicinemedicineHumansSecretionImmunology Autoimmunity and Graves' OphthalmopathyAutoantibodiesbiologyKinaseChemistryFibroblastsMiddle Agedmedicine.diseaseGraves Diseaseeye diseasesIn vitroGraves OphthalmopathyEndocrinologyThyroid Epithelial Cellsbiology.proteinFemaleThyroglobulinSignal transductionAntibodyThyroid
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SELENIUM AND AUTOIMMUNE THYROIDITIS

2023

Selenium (Se) is an essential trace element of pivotal importance to human health. Se is incorporated into selenoproteins (SePs) which have pleiotropic effects, including antioxidant and anti-inflammatory effects, and in the production of active thyroid hormone. These findings may explain the relationship between Se deficiency and pathogenesis of various human diseases including thyroid disorder. In line with these observations, the therapeutic effectiveness of Se supplementation has already been reported in patients with various thyroid diseases. However, there are still controversial data about the optimal dose of Se to be administered, as well as the duration and efficacy of treatment an…

Settore MED/49 - Scienze Tecniche Dietetiche Applicateselenium nutrition selenoproteins autoimmune thyroiditis Hashimoto’s thyroiditis Graves' disease Graves' ophthalmopathy
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